Are intrinsic inhibitory systems activated or inhibited in pathological pain states?

Behavioral and Brain Sciences 20 (3):461-462 (1997)
  Copy   BIBTEX

Abstract

Neuroplastic changes in the inhibitory systems contribute to pathological pains such as hyperalgesia. Activation or inhibition of the intrinsic inhibitory systems may depend on the pathophysiology which induces a sustained pain state. The mechanisms of hyperalgesia, opioid insensitivity following nerve injury, and opioid tolerance may be related to common neuroplastic changes. [wiesenfeld-hallin et al.]

Categories

Keywords

Reprint years

DOI

10.1017/s0140525x97461496

Other Versions

No versions found

Links

PhilArchive



    Upload a copy of this work     Papers currently archived: 100,888

External links

Setup an account with your affiliations in order to access resources via your University's proxy server

Through your library

My notes

Similar books and articles

Central inhibitory dysfunctions in neuropathic pain: What is the relationship between basic science and clinical practice?Philip J. Siddall - 1997 - Behavioral and Brain Sciences 20 (3):465-465.
Central excitation and inhibitory mechanisms and neuroplasticity are also manifested in trigeminal nociceptive pathways.James W. Hu & Barry J. Sessle - 1997 - Behavioral and Brain Sciences 20 (3):453-454.
Role of capsaicin-sensitive afferent nerves in initiation and maintenance of pathological pain.Gábor Jancsó, Mária Dux & Péter Sántha - 1997 - Behavioral and Brain Sciences 20 (3):454-455.
More inhibition and less excitation needed in the fight against pain.Rob W. Clarke - 1997 - Behavioral and Brain Sciences 20 (3):443-444.
Plasticity: Implications for opioid and other pharmacological interventions in specific pain states.Anthony H. Dickenson - 1997 - Behavioral and Brain Sciences 20 (3):392-403.
What exactly is central to the role of central neuroplasticity in persistent pain?Terence J. Coderre & Joel Katz - 1997 - Behavioral and Brain Sciences 20 (3):483-486.
Does central nervous system plasticity contribute to hyperalgesia?Corey L. Cleland & G. F. Gebhart - 1997 - Behavioral and Brain Sciences 20 (3):444-445.
Physiological antagonism between endogenous CCK and opioid: Clinical perspectives in the management of pain.Florence Noble, Rafaël Maldonado & Bernard P. Roques - 1997 - Behavioral and Brain Sciences 20 (3):460-461.
Non-stereoselective reversal of neuropathic pain by naloxone and naltrexone: involvement of toll-like receptor 4.M. Hutchinson, Y. Zhang, K. Brown, B. Coats, M. Shridhar, P. Sholar, S. Patel, N. Crysdale, J. Harrison, S. Maier, K. Rice & L. Watkins - 2008 - European Journal of Neuroscience 28 (1):20-29.
Pains are in the head, not the spine.Valerie Gray Hardcastle - 1997 - Behavioral and Brain Sciences 20 (3):451-452.

Analytics

Added to PP
2009-01-28

Downloads
19 (#1,074,273)

6 months
11 (#343,210)

Historical graph of downloads
How can I increase my downloads?

Citations of this work

No citations found.

Add more citations

References found in this work

No references found.

Add more references