We propose a theory of events and causes against the background of branching time. Notions discussed include possibility based on reality, transitions, events, determinacy, contingency, causes and effects. The main idea in defining causal relations is to introduce a certain preconditioning circumstance under which one event follows another. We also briefly compare this theory with some other theories.

Concerns about ‘mental causation’ are concerns about how it is possible for mental states to cause anything to happen. How does what we believe, want, see, feel, hope, or dread manage to cause us to act? Certain positions on the mind-body problem—including some forms of physicalism—make such causation look highly problematic. This entry sketches several of the main reasons to worry, and raises some questions for further investigation.

In her recent paper, “A Defense of Substance Causation,” Ann Whittle makes a case for substance causation. In this paper, assuming that causation is a generative or productive relation, I argue that Whittle’s argument is not successful. While substances are causally relevant in causal processes owing to outcomes being counterfactually dependent upon their role in such occurrences, the real productive work in causal processes is accomplished by the causal powers of substances.

The primary aim of this paper is to analyze the concept of degrees of causal contribution for actual events and examine the way in which it can be formally defined. This should go some way to filling out a gap in the legal and philosophical literature on causation. By adopting the conception of a cause as a necessary element of a sufficient set (the so-called NESS test) we show that the concept of degrees of causation can be given clear and (...) even empirical meaning. We then apply a game theoretical framework to derive a measure of causal contribution. Our favoured measure turns out to be a generalised version of the normalized Penrose-Banzhaf index of voting power. (shrink)

The conservation of energy and momentum have been viewed as undermining Cartesian mental causation since the 1690s. Modern discussions of the topic tend to use mid-nineteenth century physics, neglecting both locality and Noether’s theorem and its converse. The relevance of General Relativity has rarely been considered. But a few authors have proposed that the non-localizability of gravitational energy and consequent lack of physically meaningful local conservation laws answers the conservation objection to mental causation: conservation already fails in GR, so there (...) is nothing for minds to violate. This paper is motivated by two ideas. First, one might take seriously the fact that GR formally has an infinity of rigid symmetries of the action and hence, by Noether’s first theorem, an infinity of conserved energies-momenta. Second, Sean Carroll has asked how one should modify the Dirac–Maxwell–Einstein equations to describe mental causation. This paper uses the generalized Bianchi identities to show that General Relativity tends to exclude, not facilitate, such Cartesian mental causation. In the simplest case, Cartesian mental influence must be spatio-temporally constant, and hence 0. The difficulty may diminish for more complicated models. Its persuasiveness is also affected by larger world-view considerations. The new general relativistic objection provides some support for realism about gravitational energy-momentum in GR. Such realism also might help to answer an objection to theories of causation involving conserved quantities, because energies-momenta would be conserved even in GR. (shrink)

I criticize some arguments against the causal interpretability of population genetics put forward by Denis Walsh ([2007], [2010]). In particular, I seek to undermine the contention that population genetics exhibits frame of reference relativity or subjectivity with respect to its formal representations. I also show that classical population genetics does not fall foul of some criteria for causal representation put forward by James Woodward ([2003]), although those criteria do undermine some causalist stances. 1 Introduction2 Modularity3 The Crucially Important Point4 The (...) Gillespie Case: Density-Dependent Selection5 Conclusion. (shrink)

We provide an introduction to some of the key issues raised in this volume by considering how individual chapters bear on the prospects of what may be called a ‘counterfactual process view’ of causal reasoning. According to such a view, counterfactual thought is an essential part of the processing involved in making causal judgements, at least in a central range of cases that are critical to a subject’s understanding of what it is for one thing to cause another. We argue (...) that one fruitful way of approaching the different contributions to the volume is to think of them as providing materials, conceptual as well as empirical, for challenging counterfactual process views of causal thinking, or for responding to such challenges. Amongst the challenges we consider are ones that arise out of or parallel objections to counterfactual theories of causation in philosophy, or ones that appeal to apparent developmental dissociations between causal and counterfactual reasoning abilities. Possible responses turn on questions such as the following: What should count as engaging in counterfactual reasoning? How should we think of the cognitive prerequisites of such reasoning? Is it right to ask what the relationship is between causal and counterfactual reasoning, or are there in fact a number of different ways in which the two are connected? (shrink)

The counterfactual analysis of causation has focused on one particular counterfactual conditional, taking as its starting-point the suggestion that C causes E iff (C E). In this paper, some consequences are explored of reversing this counterfactual, and developing an account starting with the idea that C causes E iff (E C). This suggestion is discussed in relation to the problem of pre-emption. It is found that the 'reversed' counterfactual analysis can handle even the most difficult cases of pre-emption with only (...) minimal complications. The paper closes with a discussion of the wider philosophical implications of developing a reversed counterfactual analysis, especially concerning the differentiation of causes from causal conditions, causation by absences, and the extent to which causes suffice for their effects. (shrink)

According to proponents of the causal exclusion problem, there cannot be a sufficient physical cause and a distinct mental cause of the same piece of behaviour. Increasingly, the causal exclusion problem is circumvented via this compatibilist reasoning: a sufficient physical cause of the behavioural effect necessitates the mental cause of the behavioural effect, so the effect has a sufficient physical cause and a mental cause as well. In this paper, I argue that this compatibilist reply fails to resolve the causal (...) exclusion problem. (shrink)

We argue in favor of merely disjunctive effects, namely cases in which an event or fact, C, is not a cause of an effect, E1, and is also not a cause of a distinct effect, E2, and yet C is a cause of the disjunctive effect (E1 orE2). Disjunctive effects let us retain the additivity and the distributivity of causation. According to additivity, if C is a cause of E1 and C is a cause of E2, then C is a (...) cause of E1 and E2. According to distributivity, if C is a cause of E1 and E2, then C is a cause of E1 and C is a cause of E2. We draw an analogy between causation and intensional notions like believing, wanting, and owing, which also admit of merely disjunctive cases. We argue that both the Lewisian counterfactual account of causation (including its recent emendation by Sartorio) and the contrastive account of causation fail to properly account for this phenomenon. (shrink)

Rom Harré criticizes critical realism for ascribing causal powers to social structures, arguing that it is human individuals, and not social structures, that possess causal powers, and that a false conception of structural causation undermines the emancipatory potential of critical realism. I argue that an interpretation of the category of process as the spatio-temporalization of the category of structure, which underpins much evolutionary theory, provides the conceptual tools to explain how the critical realist transformational model of social activity can escape (...) from Harré’s criticism, leading to a general conception of social development within which various types of evolutionary processes can be identified as particular cases. I then argue that Tony Lawson’s PVRS model provides an evolutionary perspective that enables the conceptualization of coercive power as selective pressure. (shrink)

Larry Wright and others have advanced causal accounts of functional explanation, designed to alleviate fears about the legitimacy of such explanations. These analyses take functional explanations to describe second order causal relations. These second order relations are conceptually puzzling. I present an account of second order causation from within the framework of Eells' probabilistic theory of causation; the account makes use of the population-relativity of causation that is built into this theory.

Many issues at the heart of the philosophy of action and of philosophical work on free will are framed partly in terms of causation. The leading approach to understanding both the nature of action and the explanation or production of actions emphasizes causation. What may be termed standardcausalism is the conjunction of the following two theses: firstly, an event's being an action depends on how it was caused; and secondly, proper explanations of actions are causal explanations. Important questions debated in (...) the literature on free will include: is an action's being deterministically caused incompatible with its being freely performed? Are actions free only if they are indeterministically caused? Does the indeterministic causation of an action preclude its being freely performed? Does free action require agent causation? This article concentrates on issues about action and free will that centrally involve causation. (shrink)

Whatever you think of Bertrand Russell’s famous claim that the ‘law of causality’ is (at least as of 1912) redundant in the ‘advanced sciences’, it is nonethele.

This is the first instalment of a two-part paper on the counterfactual theory of causation. It is well known that this theory is ridden with counterexamples. Specifically, the following four features of the theory suffer from problems: it understands causation as a relation between events; counterfactual dependence is understood using a metric of similarity among possible worlds; it defines a non-discriminatory concept of causation; and it understands causation as transitive. A number of philosophers have recently proposed that causation is contrastive (...) because making contrasts explicit defuses counterexamples. A contrastive causal claim has the following form: C rather than C* causes E rather than E*, where C* and E* are alternative or contrast events. In this paper, I show that making contrasts explicit does indeed defuse some counterexamples. However, I also argue that the examples discussed in the literature all share a common feature, viz. that the original causal judgement is ambiguous in one way or another. Contrasting does not help with counterexamples that do not have this feature. Part II of this paper then takes up the hard cases. (shrink)

Dynamical systems theory (DST) is gaining popularity in cognitive science and philosophy of mind. Recently several authors (e.g. J.A.S. Kelso, 1995; A. Juarrero, 1999; F. Varela and E. Thompson, 2001) offered a DST approach to mental causation as an alternative for models of mental causation in the line of Jaegwon Kim (e.g. 1998). They claim that some dynamical systems exhibit a form of global to local determination or downward causation in that the large-scale, global activity of the system governs or (...) constrains local interactions. This form of downward causation is the key to the DST model of mental causation. In this paper I evaluate the DST approach to mental causation. I will argue that the main problem for current DST approaches to mental causation is that they lack a clear metaphysics. I propose one metaphysical framework (Gillett, 2002a/b/c) that might deal with this deficiency. (shrink)

While it is generally agreed that the concept of homology refers to individuated traits that have been inherited from common ancestry, we still lack an adequate account of trait individuation or inheritance. Here I propose that we utilize a counterfactual criterion of causation to link each trait with a developmental-causal (DC) gene. A DC gene is made up of the genetic information (which might or might not be physically contiguous in the genome) that is needed for the production of the (...) organismic attributes that comprise the trait. I argue that individuated traits—phenes—correspond to organismic features that are caused by DC genes. Using such an approach, we can define a DC map, which shows the relations between each pair of phenes and provides a succinct summary of genotype-phenotype relationships and phenotypic complexity. Phenes in parents and offspring are judged to be homologous if their DC genes are composed of orthologous genetic factors. When comparing more distantly related organisms, traits are homologous when linked by a chain of parent-offspring homologs along the path of ancestry that links the two organisms. There are three possible ways to deal with the potential for multiple equivalent DC genes: maximal, minimal, and consensus homology. Whereas maximal homology has limited utility, the other two approaches have value and can help to guide research at the intersection of evolution and development. (shrink)

Volume 28, Issue 4, July 2020, Page 845-847.

Causation in Physics demonstrates the importance of causation in the physical world. It details why causal mastery of natural phenomena is an important part of the effective strategies of experimental physicists. It develops three novel arguments for the viewpoint that causation is indispensable to the ontology of some of our best physical theories. All three arguments make much of the successes of experimental physics.

This book covers the overlap between informatics, computer science, philosophy of causation, and causal inference in epidemiology and population health research. Key concepts covered include how data are generated and interpreted, and how and why concepts in health informatics and the philosophy of science should be integrated in a systems-thinking approach. Furthermore, a formal epistemology for the health sciences and public health is suggested. -/- Causation in Population Health Informatics and Data Science provides a detailed guide of the latest thinking (...) on causal inference in population health informatics. It is therefore a critical resource for all informaticians and epidemiologists interested in the potential benefits of utilising a systems-based approach to causal inference in health informatics. (shrink)

a review of Tooley's Causation: a realist approach*, with emphasis on his use of probability and Ramsey sentences.

Many models of evolution are implicitly causal processes. Features such as causal feedback between evolutionary variables and evolutionary processes acting at multiple levels, though, mean that conventional causal models miss important phenomena. We develop here a general theoretical framework for analyzing evolutionary processes drawing on recent approaches to causal modeling developed in the machine-learning literature, which have extended Pearls do-calculus to incorporate cyclic causal interactions and multilevel causation. We also develop information-theoretic notions necessary to analyze causal information dynamics in our (...) framework, introducing a causal generalization of the Partial Information Decomposition framework. We show how our causal framework helps to clarify conceptual issues in the contexts of complex trait analysis and cancer genetics, including assigning variation in an observed trait to genetic, epigenetic and environmental sources in the presence of epigenetic and environmental feedback processes, and variation in fitness to mutation processes in cancer using a multilevel causal model respectively, as well as relating causally-induced to observed variation in these variables via information theoretic bounds. In the process, we introduce a general class of multilevel causal evolutionary processes which connect evolutionary processes at multiple levels via coarse-graining relationships. Further, we show how a range of fitness models can be formulated in our framework, as well as a causal analog of Prices equation, clarifying the relationships between realized/probabilistic fitness and direct/indirect selection. Finally, we consider the potential relevance of our framework to foundational issues in biology and evolution, including supervenience, multilevel selection and individuality. Particularly, we argue that our class of multilevel causal evolutionary processes, in conjunction with a minimum description length principle, provides a conceptual framework in which identification of multiple levels of selection may be reduced to a model selection problem. (shrink)

Martha Klein; VI*—Externalism, Content and Causation1, Proceedings of the Aristotelian Society, Volume 96, Issue 1, 1 June 1996, Pages 159–176, https://doi.org/.

The two traditional Indian Buddhist philosophers – the Mādhyamika Nāgārjuna (c.150–250) and the Sautrāntika-Vasubandhu (c. 350–430) – agree that mental causation involves a causal relationship between successive consciousness moments in which the previous moments are causes and the latter moments effects. In this chapter, I investigate the nature of this relation at stake. Is it a type of relationship that requires (1) necessary connection between successive consciousness moments in which there is an internal causal connection between the previous and the (...) latter conscious moments? (2) Alternatively, is the causal relationship between consciousness moments just a matter of constant conjunctions of intrinsic natures (svabhāvas) without any necessary causal relation? If the former, the two Buddhist philosophers need to explain which of the two truths – conventional truth or ultimate truth – does this necessary relation belong and why? If the latter, they need to explain how mental causation – a continuum of interrupted mental events in which the previous moment are causes and the subsequent moments effects – possible with intrinsic natures, absent any necessary causal connection between them? Either way, the Buddhist philosophers must address these two questions concerning causal relation adequately. (shrink)