Here is a simple counterexample to David Lewis’s causal influence account of causation, one that is especially illuminating due to its connection to what Lewis himself writes: it is a variant of his trumping example.

I propose an amendment of Lewis's counterfactual analysis of causation, designed to overcome some difficulties concerning redundant causation.

In this paper I present a metaphysically minimalist but theoretically strong version of fact causation, in which the causal relata constitute a full Boolean algebra, mirroring the entailment relation of the sentences that express them. I suggest a generalization of the notion of multiple realizability of causes in terms of specificity of facts, and employ this in an interpretation of what goes on in cases of apparently redundant causation.

Backtracking influence is influence that zigzags in time. For example, backtracking influence exists when an event E_1 makes an event E_2 more likely by way of a nomic connection that goes from E_1 back in time to an event C and then forward in time to E_2. I contend that in our local region of spacetime, at least, backtracking influence is redundant in the sense that any existing backtracking influence exerted by E_1 on E_2 is equivalent to E_1's temporally (...) direct influence on E_2. I prove the redundancy of backtracking influence using several plausible physical principles without assuming any fundamental temporal or causal asymmetry. This explanation can play a prominent role in an account of why causation appears to be objectively asymmetric regardless of whether the fundamental laws are symmetric. (shrink)

The main line of Lewis's account of causation is in terms of chains of counterfactual dependence. According to his original account, a causal chain is a sequence of two or more events, with counterfactual dependence at each step; and one event is a cause of another if there is a causal chain from one to the other. But some awkward cases involving redundant causation lead him to introduce the notion of quasi-dependence. Laurie Paul has suggested a way (...) of dealing with one important class of these cases in terms of dependence proper. I shall suggest a different way of dealing with this class of cases; I shall also suggest that it is possible to deal with other awkward cases too in terms of dependence proper. (shrink)

Causation is at once familiar and mysterious. Neither common sense nor extensive philosophical debate has led us to anything like agreement on the correct analysis of the concept of causation, or an account of the metaphysical nature of the causal relation. Causation: A User's Guide cuts a clear path through this confusing but vital landscape. L. A. Paul and Ned Hall guide the reader through the most important philosophical treatments of causation, negotiating the terrain by taking (...) a set of examples as landmarks. They clarify the central themes of the debate about causation, and cover questions about causation involving omissions or absences, preemption and other species of redundant causation, and the possibility that causation is not transitive. Along the way, Paul and Hall examine several contemporary proposals for analyzing the nature of causation and assess their merits and overall methodological cogency.The book is designed to be of value both to trained specialists and those coming to the problem of causation for the first time. It provides the reader with a broad and sophisticated view of the metaphysics of the causal relation. (shrink)

Quantum mechanics portrays the universe as involving non-local influences that are difficult to reconcile with relativity theory. By postulating backward causation, retro-causal interpretations of quantum mechanics could circumvent these influences and accordingly reconcile quantum mechanics with relativity. The postulation of backward causation poses various challenges for the retro-causal interpretations of quantum mechanics and for the existing conceptual frameworks for analyzing counterfactual dependence, causation and causal explanation. In this chapter, we analyze the nature of time, causation and (...) explanation in a local, deterministic retro-causal interpretation of quantum mechanics that is inspired by Bohmian mechanics. This interpretation of quantum mechanics offers a deterministic, local ‘hidden-variables’ model of the Einstein-Podolsky-Rosen experiment that poses a new challenge for Reichenbach’s principle of the common cause. In this model, the common cause – the state of particles at the emission from the source – screens off the correlation between its effects – the distant measurement outcomes – but nevertheless fails to explain the correlation between the effects. (shrink)

The paper builds on the basically Humean idea that A is a cause of B iff A and B both occur, A precedes B, and A raises the metaphysical or epistemic status of B given the obtaining circumstances. It argues that in pursuit of a theory of deterministic causation this ‘status raising’ is best explicated not in regularity or counterfactual terms, but in terms of ranking functions. On this basis, it constructs a rigorous theory of deterministic causation that (...) successfully deals with cases of overdetermination and pre-emption. It finally indicates how the account's profound epistemic relativization induced by ranking theory can be undone. Introduction Variables, propositions, time Induction first Causation Redundant causation Objectivization. (shrink)

Nancy Cartwright begins her recent book, Hunting Causes and Using Them, by noting that while a few years ago real causal claims were in dispute, nowadays “causality is back, and with a vengeance.” In the case of the social sciences, Keith Morrison writes that “Social science asks ‘why?’. Detecting causality or its corollary—prediction—is the jewel in the crown of social science research.” With respect to the health sciences, Judea Pearl writes that the “research questions that motivate most studies in the (...) health sciences are causal in nature.” However, not all data used by people interested in making causal claims come from experiments that use random assignment to control and treatment groups. Indeed, much research in the social and health science depends on non-experimental, observational data. Thus, one of the most important problems in the social and health sciences concerns making warranted causal claims using non-experimental, observational data; viz., “Can observational data be used to make etiological inferences leading to warranted causal claims?” This paper examines one method of warranting causal claims that is especially widespread in epidemiology and the health sciences generally—the use of causal criteria. It is argued that cases of complex causation generally, and redundant causation—both causal overdetermination and causal preemption—specifically, undermine the use of such criteria to warrant causal claims. (shrink)

A key test of any philosophical account of the mind is its treatment of mental causation. Proponents of the token-identity theory point to its strengths in both “demystifying” mental causation — by identifying mental causes with the physical causal mechanisms underlying bodily movements — and in avoiding commitment to dubious forms of causal overdetermination. I argue against this account of mental causation, pointing out that it mistakenly identifies actions with bodily movements. I suggest instead treating action explanations (...) as explanations of redundant causalities in behavior, and the mental causes cited in such explanations as structural causes. (shrink)

In recent years, a number of philosophers have attempted to fix paradoxes of the counterfactual account of causation by making causation contrastive. In this framework, causation is understood to be not a two-place relationship between a cause and an effect but a three or four-place relationship between a cause, an effect and a contrast on the side of the cause, the effect or both. I argue that contrasting helps resolving certain paradoxes only if an account of admissibility (...) of the chosen set of contrasts is given. I show by means of numerous examples that it is contextual features that determine admissibility. This way, context becomes part of the semantics of causation. I finally argue that once contextualised, explicit contrasting is redundant: causation is therefore a three-place relationship between a cause, an effect and a context. (shrink)

David Lewis’s latest theory of causation defines the causal link in terms of the relation of influence between events. It turns out, however, that one event’s influencing another is neither a necessary nor sufficient condition for its being a cause of that event. In the article one particular case of causality without influence is presented and developed. This case not only serves as a counterexample to Lewis’s influence theory, but also threatens earlier counterfactual analyses of causation by admitting (...) a particularly troublesome type of preemption. The conclusion of the article is that Lewis’s influence method of solving the preemption problem fails, and that we need a new and fresh approach to the cases of redundant causation if we want to hold on to the counterfactual analysis of causation. (shrink)

In many biological experiments, due to gene-redundancy or distributed backup mechanisms, there are no visible effects on the functionality of the organism when a gene is knocked out or down. In such cases there is apparently no counterfactual dependence between the gene and the phenotype in question, although intuitively the gene is causally relevant. Due to relativity of causal relations to causal models, we suggest that such cases can be handled by changing the resolution of the causal model that represents (...) the system. By decreasing the resolution of our causal model, counterfactual dependencies can be established at a higher level of abstraction. By increasing the resolution, stepwise causal dependencies of the right kind can serve as a sufficient condition for causal relevance. Finally, we discuss how introducing a temporal dimension in causal models can account for causation in cases of non-modular systems dynamics. (shrink)

Our ordinary causal concept seems to fit poorly with how our best physics describes the world. We think of causation as a time-asymmetric dependence relation between relatively local events. Yet fundamental physics describes the world in terms of dynamical laws that are, possible small exceptions aside, time symmetric and that relate global time slices. My goal in this paper is to show why we are successful at using local, time-asymmetric models in causal explanations despite this apparent mismatch with fundamental (...) physics. In particular, I will argue that there is an important connection between time asymmetry and locality, namely: understanding the locality of our causal models is the key to understanding why the physical time asymmetries in our universe give rise to time asymmetry in causal explanation. My theory thus provides a unified account of why causation is local and time asymmetric and thereby enables a reply to Russell’s famous attack on causation. (shrink)

I argue that the Exclusion Argument against mental causation is self‐defeating and should be rejected by everyone. A new version of the Argument—the ‘Micro Exclusion Argument’—does not undermine itself, but, I argue, we should reject that argument in light of the rejection of microphysical closure defended earlier in the book. And, among other things, I present an argument against free will, an argument that presupposes incompatibilism. I then show that those who believe in non‐redundant mental causation of (...) the sort I defend in Ch. 4 can resist that argument. And so, I suggest, incompatibilists who believe in free will should embrace my most controversial claims about mental causation. (shrink)

Causes always seem to come prior to their effects. What might explain this asymmetry? Causation's temporal asymmetry isn't straightforwardly due to a temporal asymmetry in the laws of nature—the laws are, by and large, temporally symmetric. Nor does the asymmetry appear due to an asymmetry in time itself. This Element examines recent empirical attempts to explain the temporal asymmetry of causation: statistical mechanical accounts, agency accounts and fork asymmetry accounts. None of these accounts are complete yet and a (...) full explanation of the temporal asymmetry of causation will likely require contributions from all three programs. (shrink)

We raise and attempt to resolve the charge that the third noble truth of the core Buddhist teachings is redundant. If the second noble truth asserts a causal relation between craving and suffering, and if James Woodward’s interventionist account of causation is correct, we argue that these premises are sufficient to entail the difference-making described by the third noble truth. Thus, the third noble truth would be superfluous insofar as it merely makes explicit what must be the case, (...) according to a plausible theory of causation, if the second noble truth is true. Having raised the possibility that the third noble truth is redundant, we consider five possible responses to the charge, the last of which we endorse. Even if the redundancy charge is found not to stick, we believe an investigation into why this is so highlights features of the third noble truth that might be suppressed under standard glosses of its meaning. (shrink)

The overdetermination problem has long been raised as a challenge to nonreductive physicalism. Nonreductive physicalists have, in various ways, tried to resolve the problem through appeal to counterfactuals. This essay does two things. First, it takes up the question whether counterfactuals can yield an appropriate notion of causal redundancy and argues for a negative answer. Second, it examines how this issue bears on the mental causation debate. In particular, it considers the argument that the overdetermination problem simply does not (...) arise on a dependency conception of causation and shows why this idea, though initially appealing, does not address the real problem. As the essay shows, the idea derives its spurious plausibility from the fact that the dependency conception cannot even make sense of our pretheoretic idea of causal redundancy. The essay concludes by briefly discussing a possible picture of mental causation that suggests itself in light of these results. (shrink)

Time-symmetric interpretations of quantum theory are often presented as featuring "retrocausal" effects in addition to the usual forward notion of causation. This paper examines the ontological implications of certain time- symmetric theories, and finds that no dynamical notion of causation applies to them, either forward or backward. It is concluded that such theories actually describe a static picture, in which the notion of causation is relegated to a descriptor of static relationships among events. In addition, these theories (...) lead to an epistemic rather than ontologically referring, realist view of quantum states. (shrink)

permits a sound and rigorously definable notion of ‘originating cause’ or causa causans—a type of transition event—of an outcome event. Mackie has famously suggested that causes form a family of ‘inus’ conditions, where an inus condition is ‘an insufficient but non-redundant part of an unnecessary but sufficient condition’. In this essay the needed concepts of BST theory are developed in detail, and it is then proved that the causae causantes of a given outcome event have exactly the structure of (...) a set of Mackie inus conditions. The proof requires the assumption that there is no EPR-like ‘funny business’. This seems enough to constitute a theory of ‘causation’ in at least one of its many senses. Introduction The cement of the universe Preliminaries 3.1 First definitions and postulates 3.2 Ontology: propositions 3.3 Ontology: initial events 3.4 Ontology: outcome events 3.5 Ontology: transition events 3.6 Propositional language applied to events Causae causantes 4.1 Causae causantes are basic primary transition events 4.2 Causae causantes of an outcome chain 4.3 No funny business Causae causantes and inns and inus conditions 5.1 Inns conditions of outcome chains: not quite 5.2 Inns conditions of outcome chains 5.3 Inns conditions of scattered outcome events 5.4 Inus conditions for disjunctive outcome events 5.5 Inns and inus conditions of transition events Counterfactual conditionals Appendix: Tense and modal connectives in BST. (shrink)

Fundamental physics makes no clear use of causal notions; it uses laws that operate in relevant respects in both temporal directions and that relate whole systems across times. But by relating causation to evidence, we can explain how causation fits in to a physical picture of the world and explain its temporal asymmetry. This paper takes up a deliberative approach to causation, according to which causal relations correspond to the evidential relations we need when we decide on (...) one thing in order to achieve another. Tamsin's taking her umbrella is a cause of her staying dry, for example, if and only if her deciding to take her umbrella for the sake of staying dry is adequate grounds for believing she'll stay dry. This correspondence explains why causation matters: knowledge of causal structure helps us make decisions that are evidence of outcomes we seek. The account also explains why we can control the future and not the past, and why causes come before their effects. When agents properly deliberate, their decisions can never count as evidence for any outcomes they may seek in the past. From this it follows that causal relations don't run backwards. This deliberative asymmetry is itself traced back to asymmetries of evidence and entropy, providing a new way of deriving causal asymmetry from temporally symmetric laws. (shrink)

The majority of the currently flourishing theories of actual causation are located in a broadly counterfactual framework that draws on structural equations. In order to account for cases of symmetric overdeterminiation and preemption, these theories resort to rather intricate analytical tools, most of all, to what Hitchcock has labeled explicitly nonforetracking counterfactuals. This paper introduces a regularity theoretic approach to actual causation that only employs material conditionals, standard Boolean minimization procedures, and a stability condition that regulates the behavior (...) of causal models under model expansions. Notwithstanding its lightweight analytical toolbox, this regularity theory performs at least as well as the structural equations accounts with their heavy appliances. (shrink)

I argue that there are some causal relata for which it is indeterminate whether one caused the other. Positing indeterminacy in causation helps us defend contested principles in the logic of causation and makes possible new ways of thinking about the theoretical impact of symmetric causal overdetermination. I close by discussing amendments of current theories of causation that would help explain causal indeterminacy.

This article takes stock of the regularity theory of causation. It considers three challenges to the theory: the problem of joint effects, the problems of redundant causation, and omission‐involving causation. The former is often cited as a special, and especially challenging, problem for regularity theories. But the force of this problem has been greatly overstated. The threat to regularity theories instead comes from the latter two.

I argue that there are some causal relata for which it is indeterminate whether one caused the other. Positing indeterminacy in causation helps us defend contested principles in the logic of causation and makes possible new ways of thinking about the theoretical impact of symmetric causal overdetermination. I close by discussing amendments of current theories of causation that would help explain causal indeterminacy.

The aim of this dissertation is to provide an analysis of central concepts in philosophy of science from the perspective of current molecular and developmental research. Each chapter explores the ways in which particular phenomena or discoveries in molecular biology influences our philosophical understanding of the nature of scientific knowledge. The introductory prologue draws some general connections between the various threads, which revolve around two central themes: causation and explanation. Chapter Two identifies a particular type of causal relation which (...) is widespread across the sciences, but cannot be straightforwardly accommodated by extant accounts of causation and causal explanation. Chapter Three explores how the form of redundant causality identified in the previous chapter plays an important role in causal explanation, by making the effect stable and robust. Chapter Four offers a novel perspective on the debate over biological reductionism by distinguishing between different paradigms of molecular explanation. Chapter Five provides a philosophical analysis of the so-called "Developmental Synthesis" of evolutionary and developmental biology, and suggests a general account of scientific unification grounded in the notion of explanatory relevance. Chapter Six offers an account of dispositional properties inspired by mechanisms of gene regulation, according to which dispositions are not properties of entities, but properties that describe the behavior of abstract idealized models. Finally, Chapter Seven scrutinizes the concept of the molecular ecosystem, a metaphor frequently employed by biologists to describe cellular interactions, but seldom articulated in detail. (shrink)

My dissertation concerns the long-standing mind-body problem in a contemporary context. I investigate whether a content property of a mental state can be causally efficacious in bringing about behavior. I argue that general objections against the causal efficacy of content are not warranted. I then propose my own account of the causal efficacy of content. ;In Chapter 1, I examine the claim that the supervenience thesis renders mental causation incompatible with underlying physical causation. I argue that if we (...) understand the supervenience thesis and the concept of causation probabilistically, it turns out that the causal efficacy of the mental is compatible with that of the physical. ;In Chapter 2, I claim that given the compatibility claim established in the previous chapter, the computational theory of the mind can be understood as a causal hypothesis. I examine this hypothesis in details and Fodor's "narrow content" proposal. I argue that Fodor's proposal has a serious problem. ;In Chapter 3, I discuss individualism which implies that content is not a psychologically relevant property. I argue that individualism is in conflict with legitimate scientific practices and that there is no reason not to individuate psychological properties in terms of diachronic, causal relations. ;In Chapter 4, I investigate the screening-off argument that since content is unnecessary and redundant for the genesis of behavior, content cannot be causally efficacious. I argue that the argument is not valid. I also critically examine three accounts of the causal efficacy of content and claim that they fall short of conferring causal efficacy to content. ;In Chapter 5, I propose an account of causal efficacy which is consistent with the requirement of causal transaction via local, physical mechanism. My account is based on the notion of causal connection which is, in turn, understood as a category of token probabilistic causal significances. According to my account, content can be causally efficacious in bringing about behavior. I conclude that reasons qua reasons have causal have causal efficacy. (shrink)

This paper argues that so-called “trumping preemption” is in fact overdetermination or early preemption, and is thus not a distinctive form of redundant causation. I draw a novel lesson from cases thought to be trumping: that the boundary between preemption and overdetermination should be reconsidered.

This paper defends the view that Newtonian forces are real symmetrical and non-causal relations. In the first part, I argue that Newtonian forces are real; in the second part, that they are relations; in the third part, that they are symmetrical relations; in the fourth part, that they are not causal relations, (but causal relata) by which I mean that they are not species of causation. The overall picture is anti-humean to the extent that it defends the (...) existence of forces, irreducible to spatio-temporal relations, but is still compatible with humean approaches to causation (and others) since it denies that forces are species of causation. (shrink)

Setting aside all exclusion-style worries about the redundancy of postulating additional, non-physical mental causes for effects that can already be explained in purely physical terms, dualists who treat mental properties as supervening on physical properties still face a further problem: in cases of mental-to-mental causation, they cannot avoid positing an implausibly coincidental coordination in the timing of the distinct causal processes terminating, respectively, in the mental effect and its physical base. I argue that this problem arises regardless of whether (...) one treats the mental cause as causing both the mental effect and its physical base, or whether one treats the latter as having instead been caused by some physical state, and that the problem also does not depend on which theory of causation one adopts. A third option of treating the mental cause and its physical base as producing the mental effect and its physical base by a single causal process that they both contribute to is found to come up short as well. (shrink)

We articulate the problems posed by the quantum liar experiment (QLE) for backwards causation interpretations of quantum mechanics, time-symmetric accounts and other dynamically oriented local hidden variable theories. We show that such accounts cannot save locality in the case of QLE merely by giving up “lambda-independence.” In contrast, we show that QLE poses no problems for our acausal Relational Blockworld interpretation of quantum mechanics, which invokes instead adynamical global constraints to explain Einstein–Podolsky–Rosen (EPR) correlations and QLE. We make the (...) case that the acausal and adynamical perspective is more fundamental and that dynamical entities obeying dynamical laws are emergent features grounded therein. (shrink)

Mental causation is a problem and not just a problem for the nonphysicalist. One of the many lessons learned from Jaegwon Kim’s writings in the philosophy of mind is that mental causation is a problem for the nonreductive physicalist as well. A central component of the common sense picture we have of ourselves as persons is that our beliefs and desires causally explain our actions. But the completeness of the “brain sciences” threatens this picture. If all of our (...) actions are causally explained by neurophysiological events occurring in our brains, what causal role is left for our reasons and motives? It would seem that these brain events do all the causal work there is to do, thus robbing the mental of its efficacy altogether or else making it a merely superfluous or redundant causal factor. This essay presents a systematic treatment of this exclusion dilemma from the perspective of a nonreductive physicalist. I argue that both horns of this dilemma can be avoided if we ground mental causation in counterfactual dependence between distinct events and understand the mind-body relation as event realization. Although in the final analysis our actions are overdetermined by their mental and neurophysiological antecedents, this overdetermination is entirely unproblematic. (shrink)

Whatever you think of Bertrand Russell’s famous claim that the ‘law of causality’ is (at least as of 1912) redundant in the ‘advanced sciences’, it is nonethele.

A regularity theory of causation analyses type-level causation in terms of Boolean difference-making. The essential ingredient that helps this theoretical framework overcome the problems of Hume’s and Mill’s classical accounts is a principle of non-redundancy: only Boolean dependency structures from which no elements can be eliminated track causation. The first part of this paper argues that the recent regularity theoretic literature has not consistently implemented this principle, for it disregarded an important type of redundancies: structural redundancies. Moreover, (...) it is shown that a regularity theory needs to be underwritten by a hitherto neglected metaphysical background assumption stipulating that the world's causal makeup is not ambiguous. Against that background, the second part then develops a new regularity theory that does justice to all types of redundancies and, thereby, provides the first all-inclusive notion of Boolean difference-making. (shrink)

A regularity theory of causation analyses type-level causation in terms of Boolean difference-making. The essential ingredient that helps this theoretical framework overcome the problems of Hume’s and Mill’s classical accounts is a principle of non-redundancy: only Boolean dependency structures from which no elements can be eliminated track causation. The first part of this article argues that the recent regularity-theoretic literature has not consistently implemented this principle, for it disregarded an important type of redundancies: structural redundancies. Moreover, it (...) is shown that a regularity theory needs to be underwritten by a hitherto neglected metaphysical background assumption stipulating that the world’s causal makeup is not ambiguous. Against that background, the second part then develops a new regularity theory that does justice to all types of redundancies and, thereby, provides the first all-inclusive notion of Boolean difference-making. (shrink)

In this essay, I defend a mind-body dualism, according to which human minds are immaterial substances that exercise non-redundant causal powers over bodies, against the notorious problem of psychophysical causation. I explicate and reply to three formulations of the problem: (i) the claim that, on dualism, psychophysical causation is inconsistent with physical causal closure, (ii) the claim that psychophysical causation on the dualist view is intolerably mysterious, and (iii) Jaegwon Kim’s claim that dualism fails to account (...) for causal pairings. Ultimately, I conclude that these objections fail and that dualist interactionism is no more problematic or mysterious than physical causation. (shrink)

Sydney Shoemaker has attempted to save mental causation by a new account of realization. As Brian McLaughlin argues convincingly, the account has to face two major problems. First, realization does not guarantee entailment. So even if mental properties are realized by physical properties, they need not be entailed by them. This is the first, rather general metaphysical problem. A second problem, which relates more directly to mental causation is that Shoemaker must appeal to some kind of proportionality as (...) a constraint on causation in order to avoid redundant mental causation. I argue that, in addition, a “piling problem” arises, since causal powers seem to be bestowed twice. Then, I try to sketch an alternative view of the relation between causal powers and properties—a reductionist view—which fares better on some accounts. But it may have to face another and, perhaps, serious problem, the “problem of the natural unity of properties”. Finally, I will pose a question about the relation between causal powers and causation. (shrink)

I criticize and emend J L Mackie's account of causal priority by replacing ‘fixity’ in its central clause by 'x is a causal condition of y, but y is not a causal condition of x'. This replacement works only if 'is a causal condition of' is not a symmetric relation. Even apart from our desire to account for causal priority, it is desirable to have an account of nonsymmetric conditionship. Truth, for example, is a condition of knowledge, but knowledge is (...) not a condition of truth. My definitions of 'sufficient condition for' and 'necessary condition for' do not imply that p is a sufficient condition of q if and only if q is a necessary condition of p. (shrink)

This essay enriches causal models capturing the propagation of prejudice, bias, and other aggregative social mechanisms, negative or positive. These explananda include the reinforcement of economic inequality, “mob-like” behavior, peer pressure, and the establishment of social norms. The stage is set by introducing various forms of redundant causation and discussing some difficulties with mainstream preemption. Next the main proposal extends current representations of aggregative social mechanisms in two respects. First, it is more nuanced, as it identifies three distinct (...) kinds of inferences: relevance of causes to effects, degree of redundancy of an effect, and the influence that causes exert on other causes. Second, it offers a quantitative—as opposed to merely qualitative—distinction between the causal contribution of preempting and preempted causes. (shrink)

Causal overdetermination occupies an uncomfortable place within all the major theories of causation. A natural solution to the problems it gives rise to would be to resolve overdetermination into preemption or joint causation. However, such a solution would seem to lead to individuate events in a fragile manner. The issue of such modal fragility is addressed and it is argued that events designated as effects are always fragile in a natural way and the putative problems of adopting modal (...) fragility can be resolved. It is also studied whether causal overdetermination could work in a setting where the overdetermining causes occur in different levels of reality. Typically postulated higher-level causes are shown to contradict the definitional features of overdetermination. There is thus no reasonable role for causal overdetermination and the notion should be abandoned. (shrink)

This thesis in an invistigation into the concept of "harm" and its moral relevance. A common view is that an analysis of harm should include a counterfactual condition: an act harms a person iff it makes that person worse off. A common objection to the moral relevance of harm, thus understood, is the non-identity problem. -/- This thesis criticises the counterfactual condition, argues for an alternative analysis and that harm plays two important normative roles. -/- The main ground for rejecting (...) the counterfactual condition is that it has unacceptable consequences in cases of overdetermination and pre-emption. Several modifications to the condition are considered but all fail to solve this problem. -/- According to the alternative analysis to do harm is to perform an act which is responsible for the obtaining of a state of affairs which makes a person’s life go worse. It is argued that should be understood in terms of counterfactual dependence. This claim is defended against counterexamples based on redundant causation. An analysis of is also provided using the notion of a well-being function. It is argued that by introducing this notion it is possible to analyse contributive value without making use of counterfactual comparisons and to solve the non-identity problem. -/- Regarding the normative importance of harm, a popular intuition is that there is an asymmetry in our obligations to future people: that a person would have a life worth living were she to exist is not a reason in favour of creating that person while that a person would have a life not worth living is a reason against creating that person. It is argued that the asymmetry can be classified as a moral option grounded in autonomy. Central to this defence is the suggestion that harm is relevant to understanding autonomy. Autonomy involves partly the freedom to pursue one’s own aims as long as one does no harm. (shrink)

When evaluating theories of causation, intuitions should not play a decisive role, not even intuitions in flawlessly-designed thought experiments. Indeed, no coherent theory of causation can respect the typical person’s intuitions in redundancy (pre-emption) thought experiments, without disrespecting their intuitions in threat-and-saviour (switching/short-circuit) thought experiments. I provide a deductively sound argument for these claims. Amazingly, this argument assumes absolutely nothing about the nature of causation. I also provide a second argument, whose conclusion is even stronger: the typical (...) person’s causal intuitions are thoroughly unreliable. This argument proceeds by raising the neglected question: in what respects is information about intermediate and enabling variables relevant to reliable causal judgment? (shrink)

The article discusses the limitations of psycho-physical parallellism and the implications of a pscycho-physical interaction paradigm considering the notion Downward Causation. The focus is on the notion of levels in nature and their interrelations, and it argues that the notion of rule-based systems should be considered a subcategory of rule-generating systems partly based on redundancy functions rather than rules.

Regularity theories of causation are guided by the idea that causes are collectively sufficient for their effects. Following Mackie [1974], that idea is typically refined to distinguish collections that include redundant members from those that do not. Causes must be collectively sufficient for their effects without redundancy. While Mackie was surely right that the regularity theory must distinguish collections that are in some sense minimally sufficient for an effect from those that include unnecessary hangers-on, I believe that redundancy (...) is the wrong mark of that distinction. I propose a way to develop the regularity theory without it. Instead of distinguishing minimal from mere sufficiency in terms of redundancy, we should look to the influence that events have in the world. Causes, so construed, must have just enough collective influence to ensure that an effect occurs. I argue that such an account provides a uniform solution to the problem of epiphenomena and to a pair of related problems that aris.. (shrink)

Causation appears to present us with an interpretative difficulty. While arguably a redundant relation given fundamental physics, it is nevertheless apparently pragmatically indispensable. This chapter revisits certain arguments made previously by the author for these claims with the benefit of hindsight, starting with the role of causal models in the human sciences, and attempting to explain why it is not possible to straightforwardly ground such models in fundamental physics. This suggests that further constraints, going beyond physics, are needed (...) to legitimate such models. These supplementary constraints could be reified, but that would seem to conflict with the completeness of physics. A response is to emphasize the practical role of causal talk, and the author suggests that a fictionalist approach might be worth exploring. After clarifying fictionalism as a general approach, they carry out in some detail the project of clarifying what a fictionalist attitude to causation would involve. (shrink)

This chapter considers the arguments for and against reductionism and then considers its main rival: emergence. A large part of the reductionist program is generally considered a failure. Philosophers of science have been chipping away at reductionist claims for a couple of decades now. Philosopher Jaegwon Kim has been a central figure in matters of reduction, supervenience, and emergence. He presents three related puzzles for the antireductionist: causal redundancy; downward causation and causal drainage. According to the author, reduction and (...) emergence have earned a place in science and the philosophy of science. The chapter finally presents the diagnosis of the controversy between reductionists and their foes and then briefly considers some theological ramifications. (shrink)

There are many ways of attaching two objects together: for example, they can be connected, linked, tied or bound together; and the connection, link, tie or bind can be made of chain, rope, or cement. Every one of these binding methods has been used as a metaphor for causation. What is the real significance of these metaphors? They express a commitment to a certain way of thinking about causation, summarized in the following thesis: ‘In any concrete situation, there (...) is an objective fact of the matter as to whether two events are in fact bound by the causal relation. It is the aim of philosophical inquiry to analyze this objective relation.’ Through a variety of examples, I hope to cast doubt on this seemingly innocuous thesis. The problem lies not with the word ‘objective’, but with the word ‘the’. The goal of a philosophical account of causation should not be to capture the causal relation, but rather to capture the many ways in which the events of the world can be bound together. 1 The metaphors 2 Unpacking the metaphors 3 Theories of causation 4 The two assassins 5 The birth control pills 6 The smoker-protector gene 7 The bicycle thief 8 Further examples 8.1 Indeterminism 8.2 Probability-lowering causes 8.3 Parts vs wholes 8.4 Symmetric overdetermination 8.5 Delayers 8.6 Causation by omission 8.7 Double prevention/disconnection 8.8 Preemptive prevention 8.9 Quantitative variables 9 Conclusion. (shrink)

When is a cause of a cause of an effect also a cause of that effect? The right answer is either Sometimes or Always . In favour of Always , transitivity is considered by some to be necessary for distinguishing causes from redundant non-causal events. Moreover transitivity may be motivated by an interest in an unselective notion of causation, untroubled by principles of invidious discrimination. And causal relations appear to add up like transitive relations, so that the obtaining (...) of the overarching relation is not independent of the obtaining of the intermediaries. On the other hand, in favour of Sometimes , often we seem not to treat events that are very spatiotemporally remote from an effect as its causes, even when connected to the effect in question by a chain of counterfactual or chance-raising dependence. Moreover cases of double prevention provide counterexamples to causal transitivity even over short chains. According to the argument of this paper, causation is non-transitive. Transitizing causation provides no viable account of causal redundancy. An unselective approach to causation may motivate resisting the distance counterexamples to transitivity, but it does not help with double prevention, and even makes it more intractable. The strongest point in favour of transitivity is the adding up of causal relations, and this is the point that extant non-transitizing analyses have not adequately addressed. I propose a necessary condition on causation that explains the adding up phenomenon. In doing so it also provides a unifying explanation of distance and double prevention counterexamples to transitivity. (shrink)

Objects and Persons presents an original theory about what kinds of things exist. Trenton Merricks argues that there are no non-living inanimate macrophysical objects -- no statues or rocks or chairs or stars -- because they would have no causal role over and above the causal role of their microphysical parts. Humans do exist: we have non-redundant causal powers. Along the way, Merricks has interesting things to say about mental causation, free will, and various philosophical puzzles. Anyone working (...) in metaphysics will enjoy this lucid and provocative book. (shrink)

In response to counterexamples involving late preemption, David Lewis (1986) revised his original (1973) counterfactual analysis of causation to include the notion of quasi-dependence. Jonardon Ganeri, Paul Noordhof and Murali Ramachandran (1998) argue that their ‘PCA*-analysis’ of causation solves the problem of late preemption and is superior to Lewis’s analysis. I show that neither quasi-dependence nor the PCA*-analysis solves the problem of late preemption.