It is widely believed that many of the competing accounts of scientific explanation have ramifications which are relevant to the scientific realism debate. I claim that the two issues are orthogonal. For definiteness, I consider Cartwright's argument that causal explanations secure belief in theoretical entities. In Section I, van Fraassen's anti-realism is reviewed; I argue that this anti-realism is, prima facie, consistent with a causal account of explanation. Section II reviews Cartwright's arguments. In Section III, it (...) is argued that causal explanations do not license the sort of inferences to theoretical entities that would embarass the anti-realist. Section IV examines the epistemic commitments involved in accepting a causal explanation. Section V presents my conclusions: contra Cartwright, the anti-realist may incorporate a causal account of explanation into his vision of science in an entirely natural way. (shrink)

Although standard Machine Learning models are optimized for making predictions about observations, more and more they are used for making predictions about the results of actions. An important goal of Explainable Artificial Intelligence (XAI) is to compensate for this mismatch by offering explanations about the predictions of an ML-model which ensure that they are reliably action-guiding. As action-guiding explanations are causal explanations, the literature on this topic is starting to embrace insights from the literature on causal models. Here (...) I take a step further down this path by formally defining the causal notions of sufficient explanations and counterfactual explanations. I show how these notions relate to (and improve upon) existing work, and motivate their adequacy by illustrating how different explanations are action-guiding under different circum- stances. Moreover, this work is the first to offer a formal definition of actual causation that is founded entirely in action-guiding explanations. Although the definitions are motivated by a focus on XAI, the analysis of causal explanation and actual causation applies in general. I also touch upon the significance of this work for fairness in AI by showing how actual causation can be used to improve the idea of path-specific counterfactual fairness. (shrink)

Not all scientific explanations work by describing causal connections between events or the world's overall causal structure. In addition, mathematicians regard some proofs as explaining why the theorems being proved do in fact hold. This book proposes new philosophical accounts of many kinds of non-causal explanations in science and mathematics.

This work consists of two parts. Part I will be a contribution to a philo- sophical discussion of the nature of causal explanation. It will present my contrastive counterfactual theory of causal explanation and show how it can be used to deal with a number of problems facing theories of causal explanation. Part II is a contribution to a discussion of the na- ture of interest explanation in social studies of science. The aim (...) is to help to resolve some controversies concerning interest explanation by explicating the concept of interest and its explanatory uses by using the account of explanation developed in Part I. (shrink)

This paper deals with the interrelationship between causal explanation and methodology in a relatively young discipline in biology: epigenetics. Based on cases from molecular and ecological epigenetics, I show that James Woodward’s interventionist account of causation captures essential features about how epigeneticists using highly diverse methods, i.e. laboratory experiments and purely observational studies, think about causal explanation. I argue that interventionism thus qualifies as a useful unifying explanatory approach when it comes to cross-methodological research efforts. It (...) can act as a guiding rationale (i) to link causal models in molecular biology with statistical models derived from observational data analysis and (ii) to identify test-criteria for reciprocal transparent studies in different fields of research, which is a shared issue across the sciences.Este artículo trata de la relación entre explicaciones causales y metodología en una disciplina biológica relativamente joven, la epigenética. Basándome en casos de la epigenética molecular y ecológica, muestro que la concepción intervencionista de la causalidad desarrollada por James Woodward capta algunos rasgos esenciales del modo en que los epigenetistas conciben la explicación causal usando métodos sumamente diversos: e.g., experimentos de laboratorio o estudios observacionales. Defiendo que el intervencionismo es útil como aproximación unificadora a la explicación cuando se trata de empresas investigadoras transdisciplinares. Puede servir como guía para (i) conectar los modelos causales en biología molecular con los modelos estadísticos derivados del análisis de datos observacionales y (ii) para identificar criterios de prueba para estudios recíprocos en diferentes ámbitos de investigación, un problema de interés común en diferentes ciencias. (shrink)

The existence of causal explanations in science has been an issue of interest in Western philosophy from its very beginnings. That is the reason this work, following an idea of Mario Bunge, makes a historical review of this matter. The modern treatment of this subject takes place since the postulation by Popper and Hempel of the D-N model of scientific explanation, whose viability is scrutinized here from different points of view in the current philosophy of science. The main (...) object of this paper is to present two arguments against the possibility of causal explanations in theoretical physics. The first one concerns the existence, in certain cases, of inter-theoretical incompatibilities, and the second refers to the need to resort, in other cases, to concatenations of laws of different theories and disciplines. The final conclusion will be the defence of a form of theoretical explanation, which follows the Popper-Hempel model, but devoid of any ontological and metaphysical connotations. (shrink)

Recent work on the interpretation of counterfactual conditionals has paid much attention to the role of causal independencies. One influential idea from the theory of Causal Bayesian Networks is that counterfactual assumptions are made by intervention on variables, leaving all of their causal non‐descendants unaffected. But intervention is not applicable across the board. For instance, backtracking counterfactuals, which involve reasoning from effects to causes, cannot proceed by intervention in the strict sense, for otherwise they would be equivalent (...) to their consequents. We discuss these and similar cases, focusing on two factors which play a role in determining whether and which causal parents of the manipulated variable are affected: Speakers' need for an explanation of the hypothesized state of affairs, and differences in the ‘resilience’ of beliefs that are independent of degrees of certainty. We describe the relevant theoretical notions in some detail and provide experimental evidence that these factors do indeed affect speakers' interpretation of counterfactuals. (shrink)

It is observed that in ordinary everyday causal explanations often just one causal factor is mentioned. One causal factor carries the explanatory burden, even if there are several causal factors that are responsible for the event to be explained. This paper deals with the question of how to account for this explanatory selection. I argue for a pragmatic stance towards explanation, that we must attend to the question–answer situation as a whole and the context of (...) the explanation. The context of an explanation includes the inquirer's and the explainer's beliefs and presuppositions relevant for the explanation-seeking question, and these are encoded in a reference class. Furthermore I argue that the explanation-giving answer contains an implicit counterfactual claim, the explanation-giving counterfactual. The solution to the problem of explanatory selection is to be found in the presuppositions encoded by the reference class and the eg-counterfactual. In short we select as explanatory that factor which, together with the presupposition encoded in the reference class we believe will make the eg-counterfactual true. (shrink)

This chapter explores the possibility of weakening the criteria for causal explanation in Making Things Happen to yield various forms of non-causal explanation. These include the following: retaining the idea that explanations must answer what if things had been different questions but dropping the requirement the answers to such questions must take the form of claims about what would happen under interventions. Retaining the w- question requirement but allowing generalizations that hold for mathematical or conceptual reasons (...) to figure in explanations. Dropping the w-question requirement to accommodate the role of information about irrelevance in explanation. (shrink)

This paper analyses the cause of disease as perceived in Classical and Hellenistic Greece, and in particular in the iamata of Epidauros, cure inscriptions collected and set up in the sanctuary of Asklepios at Epidauros in 350 BC, some of which date back to c. 450 BC. It is concluded that the iamata reflect a plurality of explanations of disease that co-existed in ancient Greece and complemented each other.

Studies exploring how students learn and understand science processes such as diffusion and natural selection typically find that students provide misconceived explanations of how the patterns of such processes arise (such as why giraffes’ necks get longer over generations, or how ink dropped into water appears to “flow”). Instead of explaining the patterns of these processes as emerging from the collective interactions of all the agents (e.g., both the water and the ink molecules), students often explain the pattern as being (...) caused by controlling agents with intentional goals, as well as express a variety of many other misconceived notions. In this article, we provide a hypothesis for what constitutes a misconceived explanation; why misconceived explanations are so prevalent, robust, and resistant to instruction; and offer one approach of how they may be overcome. In particular, we hypothesize that students misunderstand many science processes because they rely on a generalized version of narrative schemas and scripts (referred to here as a Direct-causal Schema) to interpret them. For science processes that are sequential and stage-like, such as cycles of moon, circulation of blood, stages of mitosis, and photosynthesis, a Direct-causal Schema is adequate for correct understanding. However, for science processes that are non-sequential (or emergent), such as diffusion, natural selection, osmosis, and heat flow, using a Direct Schema to understand these processes will lead to robust misconceptions. Instead, a different type of general schema may be required to interpret non-sequential processes, which we refer to as an Emergent-causal Schema. We propose that students lack this Emergent Schema and teaching it to them may help them learn and understand emergent kinds of science processes such as diffusion. Our study found that directly teaching students this Emergent Schema led to increased learning of the process of diffusion. This article presents a fine-grained characterization of each type of Schema, our instructional intervention, the successes we have achieved, and the lessons we have learned. (shrink)

The main goal of this dissertation is to provide a solid foundation for a formalization of Inference to the Best Explanation (IBE). This foundation consists of three major components. First, an intuitively adequate and formally precise model of causal explanation. Secondly, an intuitively adequate and formally precise measure of (causal) explanatory power. And third, an intuitively adequate and formally precise criterion of proportionality that is able to identify the most appropriate level of specificity for a (...) class='Hi'>causal explanation. While the first component makes it possible to generate and identify causal explanations reliably, the second and third components make it possible to evaluate the strength or quality of causal explanations, which is crucial for identifying the best of a set of competing causal explanations. (shrink)

It is widely held that it is difficult, if not impossible, to apply causal theory to the domain of quantum mechanics. However, there are several recent scientific explanations that appeal crucially to quantum processes, and which are most naturally construed as causal explanations. They come from two relatively new fields: quantum biology and quantum technology. We focus on two examples, the explanation for the optical interferometer LIGO and the explanation for the avian magneto-compass. We analyse the (...) explanation for the avian magneto-compass from the perspective of Woodward's interventionist theory and provide a causal model. Furthermore, we show how worries expressed by Woodward about quantum causation are circumvented in these cases, concluding that these kinds of explanations are most naturally construed as causal. (shrink)

We develop an account of laboratory models, which have been central to the group selection controversy. We compare arguments for group selection in nature with Darwin's arguments for natural selection to argue that laboratory models provide important grounds for causal claims about selection. Biologists get information about causes and cause-effect relationships in the laboratory because of the special role their own causal agency plays there. They can also get information about patterns of effects and antecedent conditions in nature. (...) But to argue that some cause is actually responsible in nature, they require an inference from knowledge of causes in the laboratory context and of effects in the natural context. This process, cause detection, forms the core of an analogical argument for group selection. We discuss the differing roles of mathematical and laboratory models in constructing selective explanations at the group level and apply our discussion to the units of selection controversy to distinguish between the related problems of cause determination and evaluation of evidence. Because laboratory models are at the intersection of the two problems, their study is crucial for framing a coherent theory of explanation for evolutionary biology. (shrink)

If physical reality is nonseparable, as quantum mechanics suggests, then it may contain processes of a quite novel kind. Such nonseparable processes could connect space-like separated events without violating relativity theory or any defensible locality condition. Appeal to nonseparable processes could ground theoretical explanations of such otherwise puzzling phenomena as the two-slit experiment, and EPR- type correlations. We find such phenomena puzzling because they threaten cherished conceptions of how causes operate to produce their effects. But nonseparable processes offer us an (...) alternative deal of natural order, conformity to which makes such phenomena seem quite normal and not at all unexpected. Attempts to answer the further question, as to whether an appeal to a nonseparable process provides a genuine "causal" explanation, have something to teach us about our concept of causation, but do not threaten to undermine the value of the explanation itself. (shrink)

Some causal explanations are non-committal in that mention of a property in the explanans conveys information about the causal origin of the explanandum even if the property in question plays no causal role for the explanandum . Programme explanations are a variety of non-committal causal (NCC) explanations. Yet their interest is very limited since, as I will argue in this paper, their range of applicability is in fact quite narrow. However there is at least another variety (...) of NCC explanations, causal orientation explanations, which offer a plausible model for many explanations in the special sciences. (shrink)

In the recent literature on causal and non-causal scientific explanations, there is an intuitive assumption according to which an explanation is non-causal by virtue of being abstract. In this context, to be ‘abstract’ means that the explanans in question leaves out many or almost all causal microphysical details of the target system. After motivating this assumption, we argue that the abstractness assumption, in placing the abstract and the causal character of an explanation in (...) tension, is misguided in ways that are independent of which view of causation or causal explanation one takes to be most accurate. On major accounts of causation, as well as on major accounts of causal explanation, the abstractness of an explanation is not sufficient for it being non-causal. That is, explanations are not non-causal by dint of being abstract. (shrink)

The mechanistic and causal accounts of explanation are often conflated to yield a ‘causal-mechanical’ account. This paper prizes them apart and asks: if the mechanistic account is correct, how can causal explanations be explanatory? The answer to this question varies according to how causality itself is understood. It is argued that difference-making, mechanistic, dualist and inferentialist accounts of causality all struggle to yield explanatory causal explanations, but that an epistemic account of causality is more promising (...) in this regard. (shrink)

Recent philosophical work has explored the distinction between causal and non-causal forms of explanation. In this literature, topological explanation is viewed as a clear example of the non-causal variety–it is claimed that topology lacks temporal information, which is necessary for causal structure. This paper explores the distinction between topological and causal forms of explanation and argues that this distinction is not as clear cut as the literature suggests. One reason for this is (...) that some explanations involve both topological and causal information. In these “borderline” cases scientists explain some outcome by appealing to the causal topology of the system of interest. These cases help clarify a type of topological explanation that is genuinely causal, but that differs from standard topological and interventionist accounts of explanation. (shrink)

This book aims to answer two main questions about Aristotle’s theory of causality and causal explanation, especially in relation to natural science: (1) How does he answer the main philosophical questions about causes to which he thinks his predecessors’ answers are flawed? (2) How do his answers bear on the main questions we confront in thinking about causality in general? The texts that deal with causality directly are analyzed against the background of his criticisms of his predecessors and (...) his broader views about explanation, highlighting their theoretically important commitments. This allows us to reveal the conceptual architecture of his theory. The basic theory is then applied to two questions about causality, one metaphysical and one epistemological. The metaphysical question is how to account for the relation between causes and what they cause, in light of a fundamental tension between different desiderata for causal explanations. Aristotle’s answer is pluralistic: he does not think that there is one metaphysical relationship between causes and what they cause even within a given mode of causality. The epistemological question asks how we grasp causes, and grasp them in a way that yields scientific understanding. Aristotle’s answer is again a pluralist one: metaphysically different phenomena demand different cognitive resources, and some are more accessible than others. Nevertheless, there is a discernible path from simpler to more sophisticated types of cognitive grasp on causality, in such a way that Aristotle can plausibly satisfy the epistemological constraints he thinks his predecessors cannot. Aristotle thus aims to provide a non-reductionist account of causality that still leaves room for a substantive conception of understanding in natural science. (shrink)

The book discusses recent developments in philosophy with regards to how historical events can be explained causally and introduces perspectives from the philosophy of science into the philosophy of history. The book covers the main approaches to causal explanation in history and makes clear the main issues and concepts that are associated with such explanations. For example, the book discusses historical contingency and narratives, among other issues. The book is directed to philosophers of science, philosophers of history, historians, (...) historically oriented social scientists, and everyone who is interested in how we make sense of the past. (shrink)

This paper characterizes the role of the experimenter in causal explanations of laboratory phenomena. Causal explanation rests on appeals to the experimenter's efficacy as a causal agent. I contrast "demographic" and "genetic" explanations of stochastic outcomes in a set of competition experiments in ecology. The demographic view ascribes causes to the experimenter's agency in setting up the experiment and to events within the experimental set-up. The genetic view ascribes causes to an unrecognized effect of the experimenter's (...) sampling process prior to the experimental set-up. (shrink)

The basic question of this article is whether Thomas Aquinas's doctrine of divine providence through his understanding of primary and secondary causation can be understood as a theological causal or non-causal explanation. To answer this question, I will consider some contemporary discussions about the nature of causal and non-causal explanations in philosophy of science and metaphysics, in order to integrate them into a theological discourse that appeals to the classical distinction between God as first cause (...) and creatures as secondary causes to explain God's presence and providence in the created universe. My main argument will hold that, even if there are some philosophical models of explanation that seem to allow one to suggest that, at least partially, this doctrine could be seen as a non-causal theological explanation, there are other models that offer seemingly stronger reasons to see this doctrine in full as a causal theological explanation. (shrink)

The periodic table represents and organizes all known chemical elements on the basis of their properties. While the importance of this table in chemistry is uncontroversial, the role that it plays in scientific reasoning remains heavily disputed. Many philosophers deny the explanatory role of the table and insist that it is “merely” classificatory (Shapere, in F. Suppe (Ed.) The structure of scientific theories, University of Illinois Press, Illinois, 1977; Scerri in Erkenntnis 47:229–243, 1997). In particular, it has been claimed that (...) the table does not figure in causal explanation because it “does not reveal causal structure” (Woody in Science after the practice turn in the philosophy, history, and social studies of science, Routledge Taylor & Francis Group, New York, 2014). This paper provides an analysis of what it means to say that a scientific figure reveals causal structure and it argues that the modern periodic table does just this. It also clarifies why these “merely” classificatory claims have seemed so compelling–this is because these claims often focus on the earliest periodic tables, which lack the causal structure present in modern versions. (shrink)

Carl Hempel argued that probabilistic hypotheses are limited in what they can explain. He contended that a hypothesis cannot explain why E is true if the hypothesis says that E has a probability less than 0.5. Wesley Salmon and Richard Jeffrey argued to the contrary, contending that P can explain why E is true even when P says that E’s probability is very low. This debate concerned noncontrastive explananda. Here, a view of contrastive causal explanation is described and (...) defended. It provides a new limit on what probabilistic hypotheses can explain; the limitation is that P cannot explain why E is true rather than A if P assign E a probability that is less than or equal to the probability that P assigns to A. The view entails that a true deterministic theory and a true probabilistic theory that apply to the same explanandum partition are such that the deterministic theory explains all the true contrastive propositions constructable from that partition, whereas the probabilistic theory often fails to do so. (shrink)

A defence of non-causal explanations of events is presented in cases where explanation is understood as modal explanation. In such cases the source of modal information is crucial. All explanations implicitly use contrast classes, and relative to a particular contrast we can privilege some difference makers over others. Thinking about changes in these privileged “actual” difference makers is then the source of modal information for any given explanandum. If an actual difference maker is non-causal, then we (...) have a principled definition of a non-causal explanation of an event regardless of how much causal information is also contained in the explanans. A demarcation of explanation into causal and non-causal in this way recovers ordinary language about explanation as well as reflecting genuine differences in practice, such as the in-principle evidential base for any modal claim. (shrink)

From 1959 until 1969, Heidegger lectured to psychiatrists and psychiatry students at the University of Zurich Psychiatric Clinic and in Zollikon. The transcriptions of these lectures were published as the Zollikon Seminars. In these seminars Heidegger is highly critical of psychoanalysis, because of its causal and objectifying approach to the human being. In general, Heidegger considers it an objectification or even an elimination of the human being to approach a patient from a causal perspective. In our view Heidegger (...) has overlooked the peculiar nature and complexity of psychotherapy and psychiatry, namely that psychiatry is not just a discipline that combines a hermeneutical approach and a natural science approach on a theoretical level, but it also deals with psychopathology in practice. We argue, also referring to Strawson and Gadamer, that in psychiatric practice causal explanation and hermeneutic understanding are no mutually exclusive approaches. We conclude that the encounter of philosophy and psychiatry in matters of causality and motivation could be particularly fruitful when the practical situation is addressed, recognizing the special character of psychopathology. (shrink)

This paper is an attempt to exhibit the contrast between the primitive notion of causality, simply as efficacy, and the restricted meaning of the expression "causal explanation" as associated with the application of a methodology of causal "inquiry". there can be differences of opinion on whether or not a single general methodology of causal inquiry is appropriate for natural phenomena and for human intentional actions. philosophers who reject a "methodology" of causal explanation for intentional (...) actions are not rejecting causality. the basic primitive experience of causal efficacy, of power exerted and received, is inescapable. a little attention to the phenomenology of experience can bring this out. (shrink)

Singular causal explanations cite explicitly, or may be paraphrased to cite explicitly, a particular factor as the cause of another particular factor. During recent years there has emerged a consensus account of the nature of an important feature of such explanations, the distinction between a factor regarded correctly in a given context of inquiry as ‘the cause’ of a given result and those other causally relevant factors, sometimes called ‘mere conditions’, which are not regarded correctly in that context of (...) inquiry as the cause of that result. In this paper that consensus account is characterized and developed. The developed version is then used to illuminate some recent discussions of singular causal explanations. (shrink)

This article criticizes a view about the interpretation of human action, labeled in the text as interpretivism. This view posits a sharp separation between the natural and social sciences, to the effect that the methods of the latter cannot be applied to the former. I criticize this standpoint by reconstructing a case of educational research. As I argue, the case I analyze indicates that the arguments in support of interpretivism are contradicted by what social researchers can actually achieve. I conclude (...) that the interpretivistic claims lack support and that the general separation claim appears as problematic. (shrink)

Carl Hempel (1965) argued that probabilistic hypotheses are limited in what they can explain. He contended that a hypothesis cannot explain why E is true if the hypothesis says that E has a probability less than 0.5. Wesley Salmon (1971, 1984, 1990, 1998) and Richard Jeffrey (1969) argued to the contrary, contending that P can explain why E is true even when P says that E’s probability is very low. This debate concerned noncontrastive explananda. Here, a view of contrastive (...) class='Hi'>causal explanation is described and defended. It provides a new limit on what probabilistic hypotheses can explain; the limitation is that P cannot explain why E is true rather than A if P assign E a probability that is less than or equal to the probability that P assigns to A. The view entails that a true deterministic theory and a true probabilistic theory that apply to the same explanandum partition are such that the deterministic theory explains all the true contrastive propositions constructable from that partition, whereas the probabilistic theory often fails to do so. (shrink)

This essay examines the relation between causation and causal explanation. It distinguishes two prominent roles that causes play within the sciences. On the one hand, causes may work as metaphysical posits. From this standpoint, mainstream in contemporary philosophy, causation provides the ‘raw material’ for explanation. On the other hand, causes may be conceived as explanatory postulates, theoretical hypotheses lacking any substantial ontological commitment. This unduly neglected distinction provides the conceptual resources to revisit longstanding philosophical issues, such as (...) overdetermination and causal pluralism. It also inspires a provocative reframing of Russell’s famous, if notoriously elusive, remarks on the nature of causation. (shrink)

Bell inequalities, understood as constraints between classical conditional probabilities, can be derived from a set of assumptions representing a common causal explanation of classical correlations. A similar derivation, however, is not known for Bell inequalities in algebraic quantum field theories establishing constraints for the expectation of specific linear combinations of projections in a quantum state. In the paper we address the question as to whether a ‘common causal justification’ of these non-classical Bell inequalities is possible. We will (...) show that although the classical notion of common causal explanation can readily be generalized for the non-classical case, the Bell inequalities used in quantum theories cannot be derived from these non-classical common causes. Just the opposite is true: for a set of correlations there can be given a non-classical common causal explanation even if they violate the Bell inequalities. This shows that the range of common causal explanations in the non-classical case is wider than that restricted by the Bell inequalities. (shrink)

I use a contrastive theory of causal explanation to analyze the notion of a genetic trait. The resulting definition is relational, an implication of which is that no trait is genetic always and everywhere. Rather, every trait may be either genetic or non-genetic, depending on explanatory context. I also outline some other advantages of connecting the debate to the wider causation literature, including how that yields us an account of the distinction between genetic traits and genetic dispositions.

This paper considers the problem of causal explanation in classical and statistical thermodynamics. It is argued that the irreversibility of macroscopic processes is explained in both formulations of thermodynamics in a teleological way that appeals to entropic or probabilistic consequences rather than to efficient-causal, antecedental conditions. This explanatory structure of thermodynamics is not taken to imply a teleological orientation to macroscopic processes themselves, but to reflect simply the epistemological limitations of this science, wherein consequences of heat-work asymmetries (...) are either macroscopically measurable (entropy) or calculable (probabilities), while efficient-causal relationships are obscure or indeterminable. (shrink)

The humanistic disciplines aim to offer explanations of a wide variety of phenomena. Philosophical theories of explanation have focused mostly on explanations in the natural sciences; a much discussed theory of explanation is the causal theory of explanation. Recently it has come to be recognized that the sciences sometimes offer respectable explanations that are non-causal. This paper broadens the discussion by discussing explanations that are offered in the fields of history, linguistics, literary theory, and archaeology (...) that do not seem to fit the causal theory of explanation. We conducted an exploratory survey in acclaimed humanities textbooks to find explicitly so-called explanations and analyze their nature. The survey suggests that non-causal explanations are an integral part of the humanities and that they are of distinct kinds. This paper describes three kinds that are suggested by our survey: teleological, formal, and normative explanations. We suggest that such humanistic explanations strengthen the case for explanatory pluralism. (shrink)